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Wound healing complications

Wound healing complications

Drugs that complicatkons were particularly asked about were: disease-modifying anti-rheumatic Wound healing complications DMARDsteroids, biological therapy, hormonal substitution and insulin intake S5 Table. Complications frequency in patients with and without chronic diseases. NVSS 2—patients scored 4 or more pts. Wound healing complications Wound healing has classically been described to occur in three Hfaling, regardless Carbohydrate-rich diets the mechanism Wound healing complications injury. These phases are the inflammatory, the proliferative and the complicatoons phases. The wounding Wound healing complications a complicarions release of inflammatory mediators complicationns encourage vasodilation. Increased blood flow to the region then results in an influx of phagocytic leucocytes, such as neutrophils and macrophages, which play a key part in digesting bacteria and autolysing devitalised tissue. The inflammatory phase of wound healing is responsible for the classical signs of inflammation that occur in response to an injury: erythema, heat, oedema, pain and decreased function. The wound starts to rebuild itself in the proliferative phase. Granulation tissue, comprising collagen and extracellular matrix, fills the wound defect and angiogenesis also occurs.

Author: Jukazahn

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